Drug Therapies – Medicines for Treatment of Atrial Fibrillation – A-Fib anoxic brain damage symptoms

Blood thinners are used to prevent blood clots and stroke (anticoagulants like warfarin, coumadin, jantoven; antiplatelets like aspirin, ecotrin, plavix, ticlid); or lovenox (an anticoagulant taken by injection), and heparin (used in hospitalized patients. (plavix and ticlid are antiplatelet drugs like aspirin but they are not the same or interchangeable with aspirin. If your doctor prescribes plavix or ticlid, you should not substitute aspirin for them.)

VIDEO: stroke prevention in A-fib and anticoagulant therapy. Through interviews and animations, explains how atrial fibrillation can cause stroke and why anticoagulation is so important; discussion of: warfarin (generic name), the required [monthly] monitoring, interactions anxieux synonyme with food, alcohol and other drugs: newer anticoagulants do not required regular testing. (2:01)


In general, aspirin is less effective than warfarin. 6 (for more, see faqs questions: which is better—warfarin or aspirin? Be advised that aspirin therapy can be dangerous for otherwise healthy people, “regular aspirin therapy use in healthy people increased the occurrence of serious internal bleeding by 31%, but had no significant effect on fatal heart attack or any kind of stroke.” 7

You should also get tested for variations in the CYP2C9 and VKORC1 genes which influence how you respond to warfarin (coumadin). If your doctor doesn’t provide this testing, you may want to think about getting a second opinion. These tests could save you heart problems related to over- and under-dosing of warfarin. An alternative to blood thinners

Keep in mind: leaving patients in A-fib overworks the heart and leads to remodeling and fibrosis which increase the risk of stroke. 12 mellanie true hills of stopafib.Org asks, “should we leave folks in A-fib long term, especially the non-elderly? Between the risk of heart failure, and fibrosis from long-term remodeling increasing stroke risk, could staying in A-fib long-term be a death sentence?” 13 if your doctors only prescribe rate control meds for psychology today anxiety test your A-fib, you should question them and probably get a second opinion. Categories of rate control medications

1. Calcium channel blockers prevent or slow the flow of calcium ions into smooth muscle cells such as the heart and blood vessels. Calcium channel blockers are preferred if you have heart or lung disease. Common side effects are the heart beats too slowly, constipation, low blood pressure and hypoxic and anoxic brain injury heart failure. 14“calcium channel blockers weaken the heart and can damage the liver,” according to dr. Julian whitaker in his health & healing newsletter of october 2015.

A study of nearly 3,000 women found that “women who took calcium-channel blockers for 10 years or longer had more than double the risk for breast cancer.” no risk was found for short-term use (less than 10 years). 15 calcium-channel blockers include: diltiazem (dilt, cardizem, tilazem, cartia XT) [the generic name of a medication is listed first, the brand name is in parentheses] and verapamil (calan, isoptin).

Common side effects are: the heart beats too slowly, low blood pressure, tiredness, and loss of sex-drive. 16 , 17 in many people, beta-blockers can reduce heart rate by 10 to 30 beats per minute. 18 beta-blockers like metoprolol can be particularly dangerous for older people (over 60) and cause symptoms similar to aging, 19 beta blockers also carry substantial risk for psychiatric side effects, particularly depression. When nanoxia project s midi using beta blockers, a person’s psychiatric health should be monitored carefully. 20“beta-blockers are notorious for causing impotence, fatigue, and depression,” according to dr. Julian whitaker in his health & healing newsletter of october 2015.

Carvedilol (coreg), however, targets both the beta- and alpha-adrenergic receptors on the heart muscle. “beta-blockers (like carvedilol) which target both receptors “offer the most benefit to cardiac patients.” A study in 2003 showed that carvedilol produced a greater survival rate than metoprolol. (thanks to janet brown for calling our attention to this research.)

While digoxin is a commonly used drug for rate control, it is only effective at controlling heart rate at rest, i.E., when you are in the doctor’s office. But when you leave, your heart rate may go too high. It’s not as effective at controlling heart rate during physical activity or when stressed (when the sympathetic nervous system is more active). Beta-blockers and calcium-channel blockers are generally more effective than digoxin. 24

In a 2014 study of veterans newly diagnosed with A-fib, mortality rates were higher for digoxin-treated patients than for untreated patients. (this was a large retrospective study of 122,465 definition of anxiety disorder according to dsm 5 patients mostly male, with 28,679 receiving digoxin.) A-fib patients treated with digoxin had a more than 20% increased risk of dying within about three years. 25

According to 2017 results from the ARTISTOTLE trial, patients with A-fib taking digoxin had an increased risk of death, whether or not they had heart failure. And this risk increased with higher levels of digoxin in the bloodstream (≥1.2 ng/ml). Patients not taking digoxin before the ARISTOTLE trial who began taking it over the course of the study had a 78 percent increase in the risk of death from any cause and a four-fold increased risk of sudden death after starting digoxin.

Keep in mind: in general current “antiarrhythmic” (anti irregular heart rhythm) drugs aren’t always effective and tend to have bad side effects such as pulmonary fibrosis and impaired liver function. 29‘ 30‘ 31‘ 32 they also become less effective over time, with approximately half of the patients eventually developing resistance to them. 33 up-to 50% of patients experience a recurrence of A-fib after 1-year of antiarrhythmic treatment, and up-to 85% experience a recurrence after 2-years. 34‘ 35

Procainamide (procan SR, promine, pronestyl, procanbid): slows nerve impulses in the heart and reduces the sensitivity of heart tissue. Not FDA approved for A-fib. Long-term use associated with lupus. Generally not used as a first-time drug because nanoxia deep silence 3 anthracite of bad side effects. Less effective against A-fib than the other class 1A drugs quinidine and disopyramide. 38 (class 1A drug)

Flecainide (tambocor): slows nerve impulses in the heart and makes the heart tissue less sensitive. Approved only for paroxysmal (occasional) A-fib with structurally normal heart. Normally the first drug tried on otherwise healthy patients with new A-fib. Not recommended after a heart attack or if you have a structural heart disease. (class 1C drug)

Sotalol (betapace): not recommended (conversion from A-fib to normal rhythm rate is low). Only approved in the US for ventricular arrhythmias. (class II and class III drug—a beta-blocker with antiarrhythmic effects). Should not be used in patients with severe heart failure or those with a long QT interval (see my article: understanding the EKG signal), because it may trigger a lethal cardiac arrhythmia in those patients. May anxiety attack cure natural cause severe fatigue. 40

Amiodarone (cordarone, pacerone): not FDA-approved for A-fib. Moderately effective for conversion from A-fib to normal rhythm, but onset is slow. Good rate slowing in A-fib. This is usually the last drug tried on patients because of its toxic side effects particularly in the lungs, thyroid, and liver. (class III drug but it also blocks sodium channels like a class I drug.)

Ibutilide (corvert): not for patients with low blood potassium, a prolonged QT interval (slow heart beat), or torsade de pointes (very irregular, fast ventricular heart beats). Effective in electrical cardioversion. Often used in place of electrocardioversion (33% to 49% success rate) and is generally more effective in cases of atrial flutter than in A-fib. (class III drug anxiety disorder questionnaire pdf)

The class 1 drugs quinidine, procainamide, disopyramide, flecainide, and propafenone should probably be avoided if you’ve had a heart attack or have structural heart disease. The class III drugs amiodarone, sotatol, dofetilide, and azimilide appear to be safer to use if you have structural heart disease. 41 in structurally normal hearts, class IC drugs ( flecainide and propafenone) cause less heart rhythm problems and are the least toxic. 42

For example, leon writes that he takes 100 mg of flecainide three times at intervals of twenty minutes when he has an A-fib attack. This often shortens the time of an A-fib attack. “it (the pill-in-the-pocket treatment) has changed my life in that it reduces my time in A-fib to usually a couple of hours as opposed to between 12 to 36 hours. It allows me to recover completely in a lot quicker time, because my heart hasn’t been going crazy for a day or more. And it also allows me to remain out of hospital, which has been fantastic.” (leon, E-mail: sandman_oz (at) yahoo.Com)

Marilyn writes she would take rythmol 300 mg and inderal 20 mg, wait three hours, then take inderal 20 mg, wait three hours, then again start the rythmol 300 mg and inderal 20 mg, etc. Although she daily took a 325 mg coated aspirin, during a bout nanoxia deep silence 4 micro case of A-fib she would also chew an 81 mg baby aspirin. (marilyn, E-mail: nmshook (at) sbcglobal.Net)

Another treatment strategy is to take lower doses of an antiarrhythmic med on a regular basis, then take a higher dose during an A-fib attack. A-fib patient reg writes he takes 300 mg of flecainide, and 2 hours later goes back into SR. He normally is on a loading dose of flecainide 100 mg in the morning and 50 mg in the afternoon. (email: r.J.Tooth (at) shu.Ac.Uk)